Among the many heart disease , aortic stricture is one of them . It is a heart and soul precondition where due to the narrowing of the aortal valve the heart is unable to pump out enough blood line that is required . This makes the heart to work more which could eventually lead to heart unsuccessful person . Aortic Stenosis is one of the chief rationality for sudden cardiac deaths .

Aortic Stenosis: Definition

The aorta , which is the master arterial blood vessel , transmits the blood from the heart to the body via the aortic valve . The aortal valve when constringe , intercepts the valve and obstruct the line of descent current . This narrowing is called aortic stenosis .

When the aortic valve obstructs , the nub needs to put an extra effort in pumping origin to the consistence . This would finally belittle the blood amount that is pump out from the sum and may subvert the heart muscle .

A surgical procedure is ask to interchange the aortic valve in severe cases ; failing to do so may contribute to core problems .

Aortic Stenosis

Signs and Symptoms of Aortic Stenosis

The signs and symptom of Aortic Stenosis include :

In children and infant , symptoms of Aortic Stenosis admit :

The meek or moderate aortal stricture in shaver may get forged with age . There is a high risk of bacterial endocarditis - a pump transmission .

Causes of Aortic Stenosis

aortal stricture may be congenital , but generally it matures in later life . Children suffering from aortal stricture may have other weather condition since birth . Calcific aortic stenosis is a type of aortic stenosis that mainly happen due to calcium deposits that narrow the valve . This happens to people who have unnatural aortic or bicuspidate valves . seldom , calcification develops if the patient has undergone chest radiation therapy .

Rheumatic fever is one of the other causes which evolve scarlet fever orstrep throat . However , after a period of 5 - 10 yr valve problems do not mature . In the US , creaky febricity is becoming rare . 2 % of people over 65 years get aortic stenosis . It is more frequent in men than in women .

Pathophysiology of Aortic Stenosis

A systolic pressure slope develops between the aorta and the left ventricle and resistance to systolic ejection occurs in aortal stricture . The outflow obstruction have , leads to an increase in systolic imperativeness in left heart ventricle ( LV ) . The counteractive mechanism to normalise the stress on LV wall , parallel replication of sarcomere increase LV rampart thickness , produce concentric hypertrophy . However , the chamber is not lucubrate and the diastolic compliance is reduced . finally , LV ending - diastolic press ( LVEDP ) rises , causing an increase in pulmonary capillary arterial pressures and a decrease in cardiac production . The contractility of the myocardium may lessen , chair to a decrease in cardiac output due to systolic dysfunction . This results in heart failure .

In many patients with aortal stricture , cardiac output is maintained for many long time and LV systolic function is keep up . The cardiac output may not increase during exercise , though it is normal at rest resulting in example - induced symptom . Impaired LV relaxation or decreased LV compliance may precede to diastolic disfunction .

In patients with severe aortic stenosis , the atrial condensation plays a life-sustaining use in diastolic filling of the unexpended ventricle . This may lead to kernel failure due to an unfitness to observe cardiac production . Increased LV people , increased LV systolic pressure and extend systolic ejection elevates themyocardialoxygen requirement , peculiarly in the subendocardial region . The coronary stock flow is often reduce though it may be normal for LV mass .

Thus , myocardial perfusion is compromised by a proportional psychiatrist in myocardial hairlike denseness and a diminished diastolic transmyocardial ( coronary ) perfusion slope due to upraised LV diastolic imperativeness . Hence , the subendocardium is vulnerable to underperfusion , which leave in myocardial ischaemia .

Prognosis of Aortic Stenosis

Despite the presence of severe LV outflow tract obstruction ( LVOTO ) , patients with severe aortal stricture may show no symptom for many years . symptomless patient , even with critical aortic stenosis , have an splendid protrusion for survival of the fittest , with an expected death rate of less than 1 % per year . However , 4 % of sudden cardiac destruction in asymptomatic patients with critical aortic stenosis occur . The coalesced valve country and flow slope patterns set aside better characterization of clinical outcome among asymptomatic aortic stricture patients .

Diagnosis of Aortic Stenosis

To diagnose aortic stenosis , the following tests can be contract :

Treatment for Aortic Stenosis

medicine for Aortic Stenosis : medicine are unable to reverse aortal valve stenosis . However , a sure medications may help to get sum pace and fondness rhythm in order .

Surgical Procedures for Aortic Stenosis : The patient role with decisive aortic stenosis may necessitate aortal valve replacement or reparation . Surgery is the primary intervention for Aortic Stenosis . The therapy to replace or repair aortic valve let in :

In add-on , before any medical or dental subroutine is carried out , antibiotic need to be shell out post the aortal valve replacement surgery . This would subdue the risk of contagion in the kernel tissue ( endocarditis ) .

Prevention of Aortic Stenosis:

The precautions that can prevent aortal valve stricture are :

Aortic Stenosis could make life difficult and hence it is very important to prevent it right at the root . In showcase it has already happened , abiding by the discourse protocol is very important and so is maintaining the restrictions post the surgical methods .