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Neuromyelitis optica being an autoimmune disorder with demyelination as its characteristic presents as loss of visual sensation as its chief symptoms with red of sensations and other features ofspinal corddamage . Autoimmune , for exemplar , are generally very difficult to treat because of the fact that own immune organization has started to attack , cell and if the resistant system is to be destroy then the other timeserving infections would make their way into the body and it will cause human death much before than the presuppose fourth dimension lapse in the autoimmune disorder like neuromyelitis optica .
As far as the research usable for the neuromyelitis optica is concerned and the treatment modality that have been found for it , there is no specific operative treatment available for the disorder , in fact , there is also no definitive medical discussion useable for it.[1]In fact , the operative the can prove more detrimental to the patient role it will open up the inflaming unconscious process to the body and expose more amount of antigens to one ’s own immune system which will burst out up the severity and progression of the disease . Once diagnosed , it is an incurable disease with high morbidity .
Medical discussion is only given to downgrade the saturation of disease progression and to relieve the symptom temporarily till the drug are continue . Few operative mood can only have when it is associated with some other symptom likeglaucoma , etc . If the disorderliness is link up with optical magnetic disk defects then it can be restrict with the help of optical maser treatment for the same .
Is There A Surgery For Neuromyelitis Optica?
Medical discourse is the only handling modality to be found effective to an extent in the control of neuromyelitis optica . endovenous corticosteroids are the drug of option and the first - line treatment for neuromyelitis optica . High drug synthetical corticosteroids like methylprednisolone are the most commonly used medicine in the various regimens.[2]The number by the chemical mechanism of deoxidise the inflammation to the antigens by the crushing of the immune system of rules . The proliferation of the cell of the immune system is downgrade and the answer to the antigenic stimulation is decreased . It also work by reducing the capillary permeability around the site of excitement which results in lesser polymorphonuclear lymphocytes reaching to the site and blowing off of the inflammation .
Other various antilymphocyte medications have been used in its treatment , for example , mycophenolate mofetil , Imuran , cyclosporine , amethopterin , rituximab first - line , etc . to name a few . In recent subject , mycophenolate mofetil and azathioprine have shown promise in the remission of neuromyelitis optica.[3 ]
One other therapy which could be used with quite a winner is plasmapheresis . In this therapy , the blood of the patient is taken out by a continuous working machine like the way in dialysis and is filtered for the remotion of specific antibody against the optic nerve and spinal electric cord antigens . When these antibodies get take out from the blood , the blood is infused back in the body . It not only is better than medical treatment in providing remission but also it delays the advance of the disease but the independent disadvantage is invading nature and repetitiousness of it for the continuous removal of the antibody .
Conclusion
Being an autoimmune disorder , neuromyelitis optica has no specific treatment like the other autoimmune disorders which in effect completely cure the disease . Only the advance and rigorousness can be downgrade with the utilisation of in high spirits dose corticoid in unwritten and endovenous forms . Surgical intervention is of no use in it , in fact , it can prove detrimental to the affected role because it increases the exposure of the immune system towards the causative antigen and blaze up up the immune response against it , and so it is a contraindication .
Definite medical handling is also not available but the disorderliness could be well hold in and reversion can be prevented if the early detection is potential and diagnosis is made before gamey titers of the antibodies developed against the causative antigen .
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